Raja A., Devi K.R.U., Ramalingam B., Brennan P.J. Terapevticheskiy Arkhiv, 77, 54–59 (In Russian). Immunogenetic HLA markers of chronic viral hepatits.
Popov E.A., Levitan B.N., Alekseev L.P., Pronina O.A., Suchkov S.V. Analysis of the distribution of class I HLA-antigens in patients with systemic scleroderma with regard to features of the clinical course of the disease and therapy with D-penicillamine. Pevnitskiy L.A., Guseva N.G., Sibiryakova L.G., Oskilko T.G. Autoimmune hemolytic anemia occurred prior to evident nephropathy in a patient with chronic hepatitis C virus infection: case report. Ohsawa I., Uehara Y., Hashimoto S., Endo M., Fujita T., Ohi H. New England Journal of Medicine, 343, 702–709. PhD Thesis, American University of Beirut, Lebanon. Immunological and genetical aspects of patients with hydatid disease in Lebanon. Phenotypic analysis of peripheral lymphocyte subpopulations in hydatid patients. Major tropical syndromes by body system: Systemic infections: Hydatid disease. Clinical Microbiology Reviews, 5, 248–261. Molecular and immunological diagnosis of echinococcosis. Clinical Microbiology Reviews, 17, 107–135. Biological, epidemiological, and clinical aspects of echinococcosis, a zoonosis of increasing concern. Journal of Clinical Microbiology, 18, 1021–1026. Detection and partial characterization of circulating immune complexes in hydatid disease. D’Amelio R., Pontesilli O., Palmisano L., Pezzella M., Vullo V., Delia S., De Rose F., Sorice F., Aiuti F. Medical Microbiology and Immunology, 178, 177–186. Hydatid disease: analysis of parasite antigens in circulating immune complexes and in preformed hydatid antigen-antibody complexes. D’Amelio R., De Rosa F., Pontesilli O., Dayal R., Brighouse G., Teggi A., Barnet M., Lambert P.H. The evaluation of HLA-DRB1 antigens as susceptibility markers for unilocular cystic echinococcosis in Egyptian patients. Azab M.E., Bishara S.A., Ramzy R.M.R., Oteifa N.M., El-Hoseiny L.M., Ahmed M.A. Journal of the Egyptian Society of Parasitology, 34, 183–196. Association of some HLA-DRB1 antigens with Echinococcus granulosus specific humoral immune response. Azab M.E., Bishara S.A., Helmy H., Oteifa N.M., El-Hoseiny L.M., Ramzy R.M., Ahmed M.A.
MSc Thesis, American University of Beirut, Lebanon. Hydatid disease in Lebanon as reflected by some Lebanese hospitals. Study of humoral antibodies in experimental hydatidosis of mice. Polymorphism of the HLA genetic system in Khanty population. Abramov V.Yu., Zaretskaya Yu.M., Krivtsova L.A., Otocheva L.V. Circulating immune complexes and complement C3 and C4 levels in a selected group of patients with rhinitis in Lebanon. Abdelnoor A.M., Kobeissy F., Farhat D., Hadi U. HLA-A, B, C typing of a selected group of Lebanese.
Human leukocyte antigen (HLA) class I and II frequencies in selected groups in Lebanon. Abdelnoor A.M., Abdelnoor M., Heneine W., Kobeissy F., Mansur S., Malak R., Sharara H. The joint pain reported by some of the patients might be attributed to CIC deposits. granulosus infection have been identified. In conclusion, 2 HLA alleles that associate with resistance and 1 that associates with susceptibility to E.
However, there did not appear to be a correlation between the presence of HLA-B*35 and high antibody titers, or the presence of CICs. Twenty five patients had anti-Echinococcus antibodies and 9 patients had CICs. HLA-B*35 was found to associate with susceptibility to disease (RR = 1.70, P = 0.02). However, it appeared that linkage disequilibrium did not exist between these 2 alleles (P = 0.250). HLA-B*14 and HLA-DRB1*01 appeared to associate with protection against CE (P1 = P2 = 0.007 and P1 = P2 = 0.0007, respectively). Antibody titers were determined by indirect hemagglutination (IHA) and CIC by polyethylene glycol (PEG) precipitation. Linkage disequilibrium was determined for associated alleles using SPSS 12.0 for Windows. Relative risk and P-values were determined for each allele using Statcalc (EpiInfo, Version 6). HLA profiles were determined by DNA-SSP typing. Thirty patients and 20 controls were included. The objectives of this study were to determine HLA allele-CE association in patients, and relate its presence to high anti-Echinococcus antibody titers and the presence of circulating immune complexes (CIC). Cystic echinococcosis (CE), caused by the parasite Echinococcus granulosus, is a prominent disease in Lebanon.
0 kommentar(er)
